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fuckyeahnarcotics:

This is a synovial pathology, with discreetly calcified intra-articular bodies and extrinsic bony scalloping due to chronic pressure erosion; there is no infiltration or destruction of the bone, Primary Synovial Osteochondromatosis is the most likely diagnosis; Pigmented Villonodular Synovitis is in the differential especially as on the MRI the bodies seem to be tightly packed and almost mass like, PVNS may sometimes show minor calcification on x-rays, but rarely dense calcification like this case does, sarcoma is highly unlikely due to the chronic pressure erosions

fuckyeahnarcotics:

This is a synovial pathology, with discreetly calcified intra-articular bodies and extrinsic bony scalloping due to chronic pressure erosion; there is no infiltration or destruction of the bone, Primary Synovial Osteochondromatosis is the most likely diagnosis; Pigmented Villonodular Synovitis is in the differential especially as on the MRI the bodies seem to be tightly packed and almost mass like, PVNS may sometimes show minor calcification on x-rays, but rarely dense calcification like this case does, sarcoma is highly unlikely due to the chronic pressure erosions

(via fyeahmedlab)

fyeahmedlab:

This is what a colour-enhanced image of the inner surface of your lung looks like. The hollow cavities are alveoli; this is where gas exchange occurs with the blood.

fyeahmedlab:

This is what a colour-enhanced image of the inner surface of your lung looks like. The hollow cavities are alveoli; this is where gas exchange occurs with the blood.

(Source: cdin.us)

paosoblasian:

My x-ray as of March 15th, 2012. I didn’t feel like getting back up & crutching back over to the computer to take pictures of my new ones.Still osteopenic :-/

paosoblasian:

My x-ray as of March 15th, 2012. I didn’t feel like getting back up & crutching back over to the computer to take pictures of my new ones.
Still osteopenic :-/

medicalschool:

The coagulation cascade of secondary hemostasis has two pathways which lead to fibrin formation. These are the contact activation pathway (also known as the intrinsic pathway), and the tissue factor pathway (also known as the extrinsic pathway).
Coagulation begins almost instantly after an injury to the blood vessel has damaged the endothelium lining the vessel. Exposure of the blood to proteins such as tissue factor initiates changes to blood platelets and the plasma protein fibrinogen, a clotting factor. Platelets immediately form a plug at the site of injury; this is called primary hemostasis. Secondary hemostasis occurs simultaneously: Proteins in the blood plasma, called coagulation factors or clotting factors, respond in a complex cascade to form fibrin strands, which strengthen the platelet plug.

medicalschool:

The coagulation cascade of secondary hemostasis has two pathways which lead to fibrin formation. These are the contact activation pathway (also known as the intrinsic pathway), and the tissue factor pathway (also known as the extrinsic pathway).

Coagulation begins almost instantly after an injury to the blood vessel has damaged the endothelium lining the vessel. Exposure of the blood to proteins such as tissue factor initiates changes to blood platelets and the plasma protein fibrinogen, a clotting factor. Platelets immediately form a plug at the site of injury; this is called primary hemostasis. Secondary hemostasis occurs simultaneously: Proteins in the blood plasma, called coagulation factors or clotting factors, respond in a complex cascade to form fibrin strands, which strengthen the platelet plug.

sakuratuanct:

THESE SOME GREAT REAL PICTURES OF YOUR IMMUNE CELLS. ALL OF THEM ARE CIRCULATING IN YOUR BODY AND WORKING 24/7 TO PREVENT YOU FROM GETTING INFECTION AND FALL SICK. ^^

AREN’T YOU AMAZED BY THESE? WELL, I AM.

MY NEXT EXAM PAPER: IMMUNOLOGY~ DANG~~~~

(via fyeahmedlab)

medicalschool:

Nerve cells in Huntington’s brain died off creating a larger hole, or ventricle, in the center 

medicalschool:

Nerve cells in Huntington’s brain died off creating a larger hole, or ventricle, in the center 

medicalschool:

Cerebral angiogram

medicalschool:

Cerebral angiogram

fuckyeahnarcotics:

This is a synovial pathology, with discreetly calcified intra-articular bodies and extrinsic bony scalloping due to chronic pressure erosion; there is no infiltration or destruction of the bone, Primary Synovial Osteochondromatosis is the most likely diagnosis; Pigmented Villonodular Synovitis is in the differential especially as on the MRI the bodies seem to be tightly packed and almost mass like, PVNS may sometimes show minor calcification on x-rays, but rarely dense calcification like this case does, sarcoma is highly unlikely due to the chronic pressure erosions

fuckyeahnarcotics:

This is a synovial pathology, with discreetly calcified intra-articular bodies and extrinsic bony scalloping due to chronic pressure erosion; there is no infiltration or destruction of the bone, Primary Synovial Osteochondromatosis is the most likely diagnosis; Pigmented Villonodular Synovitis is in the differential especially as on the MRI the bodies seem to be tightly packed and almost mass like, PVNS may sometimes show minor calcification on x-rays, but rarely dense calcification like this case does, sarcoma is highly unlikely due to the chronic pressure erosions

(via fyeahmedlab)

(Source: gh0stparty)

fyeahmedlab:

This is what a colour-enhanced image of the inner surface of your lung looks like. The hollow cavities are alveoli; this is where gas exchange occurs with the blood.

fyeahmedlab:

This is what a colour-enhanced image of the inner surface of your lung looks like. The hollow cavities are alveoli; this is where gas exchange occurs with the blood.

(Source: cdin.us)

paosoblasian:

My x-ray as of March 15th, 2012. I didn’t feel like getting back up & crutching back over to the computer to take pictures of my new ones.Still osteopenic :-/

paosoblasian:

My x-ray as of March 15th, 2012. I didn’t feel like getting back up & crutching back over to the computer to take pictures of my new ones.
Still osteopenic :-/

medicalschool:

The coagulation cascade of secondary hemostasis has two pathways which lead to fibrin formation. These are the contact activation pathway (also known as the intrinsic pathway), and the tissue factor pathway (also known as the extrinsic pathway).
Coagulation begins almost instantly after an injury to the blood vessel has damaged the endothelium lining the vessel. Exposure of the blood to proteins such as tissue factor initiates changes to blood platelets and the plasma protein fibrinogen, a clotting factor. Platelets immediately form a plug at the site of injury; this is called primary hemostasis. Secondary hemostasis occurs simultaneously: Proteins in the blood plasma, called coagulation factors or clotting factors, respond in a complex cascade to form fibrin strands, which strengthen the platelet plug.

medicalschool:

The coagulation cascade of secondary hemostasis has two pathways which lead to fibrin formation. These are the contact activation pathway (also known as the intrinsic pathway), and the tissue factor pathway (also known as the extrinsic pathway).

Coagulation begins almost instantly after an injury to the blood vessel has damaged the endothelium lining the vessel. Exposure of the blood to proteins such as tissue factor initiates changes to blood platelets and the plasma protein fibrinogen, a clotting factor. Platelets immediately form a plug at the site of injury; this is called primary hemostasis. Secondary hemostasis occurs simultaneously: Proteins in the blood plasma, called coagulation factors or clotting factors, respond in a complex cascade to form fibrin strands, which strengthen the platelet plug.

(Source: fyeahanatomy, via fyeahmedlab)

sakuratuanct:

THESE SOME GREAT REAL PICTURES OF YOUR IMMUNE CELLS. ALL OF THEM ARE CIRCULATING IN YOUR BODY AND WORKING 24/7 TO PREVENT YOU FROM GETTING INFECTION AND FALL SICK. ^^

AREN’T YOU AMAZED BY THESE? WELL, I AM.

MY NEXT EXAM PAPER: IMMUNOLOGY~ DANG~~~~

(via fyeahmedlab)

(Source: brobituary, via art--life)

medicalschool:

Nerve cells in Huntington’s brain died off creating a larger hole, or ventricle, in the center 

medicalschool:

Nerve cells in Huntington’s brain died off creating a larger hole, or ventricle, in the center 

medicalschool:

Healthy human brain

medicalschool:

Healthy human brain

medicalschool:

Cerebral angiogram

medicalschool:

Cerebral angiogram

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